Published in Mol Cell Biol on November 01, 1992
Cytokine-inducible expression in endothelial cells of an I kappa B alpha-like gene is regulated by NF kappa B. EMBO J (1993) 2.75
Altered gene expression in neurons during programmed cell death: identification of c-jun as necessary for neuronal apoptosis. J Cell Biol (1994) 2.75
Cell cycle-dependent variations in c-Jun and JunB phosphorylation: a role in the control of cyclin D1 expression. EMBO J (2000) 2.66
Fra-1 induces morphological transformation and increases in vitro invasiveness and motility of epithelioid adenocarcinoma cells. Mol Cell Biol (1998) 2.22
Alternative promoter usage of the Fos-responsive gene Fit-1 generates mRNA isoforms coding for either secreted or membrane-bound proteins related to the IL-1 receptor. EMBO J (1994) 2.02
The NFAT-1 DNA binding complex in activated T cells contains Fra-1 and JunB. Mol Cell Biol (1993) 1.94
Promoter specificity and biological activity of tethered AP-1 dimers. Mol Cell Biol (2002) 1.75
CCAAT/enhancer binding protein-alpha amino acid motifs with dual TBP and TFIIB binding ability co-operate to activate transcription in both yeast and mammalian cells. EMBO J (1995) 1.70
Structure, mapping, and expression of erp, a growth factor-inducible gene encoding a nontransmembrane protein tyrosine phosphatase, and effect of ERP on cell growth. Mol Cell Biol (1993) 1.62
The Mos/MAP kinase pathway stabilizes c-Fos by phosphorylation and augments its transforming activity in NIH 3T3 cells. EMBO J (1995) 1.55
Periodic cdc25C transcription is mediated by a novel cell cycle-regulated repressor element (CDE). EMBO J (1995) 1.54
Transcriptional activation of the fra-1 gene by AP-1 is mediated by regulatory sequences in the first intron. Mol Cell Biol (1995) 1.47
c-Fos-induced activation of a TATA-box-containing promoter involves direct contact with TATA-box-binding protein. Mol Cell Biol (1994) 1.42
The repertoire of fos and jun proteins expressed during the G1 phase of the cell cycle is determined by the duration of mitogen-activated protein kinase activation. Mol Cell Biol (1999) 1.39
The human papillomavirus type 16 E7 gene product interacts with and trans-activates the AP1 family of transcription factors. EMBO J (1996) 1.33
Defective dorsal closure and loss of epidermal decapentaplegic expression in Drosophila fos mutants. EMBO J (1997) 1.31
Activation of the granulocyte-macrophage colony-stimulating factor promoter in T cells requires cooperative binding of Elf-1 and AP-1 transcription factors. Mol Cell Biol (1994) 1.28
Proliferative activation of quiescent Rat-1A cells by delta FosB. Mol Cell Biol (1993) 1.27
Cell transformation by c-fos requires an extended period of expression and is independent of the cell cycle. Mol Cell Biol (1994) 1.22
Accumulation of Fra-1 in ras-transformed cells depends on both transcriptional autoregulation and MEK-dependent posttranslational stabilization. Mol Cell Biol (2003) 1.18
Ubiquitin-independent proteasomal degradation of Fra-1 is antagonized by Erk1/2 pathway-mediated phosphorylation of a unique C-terminal destabilizer. Mol Cell Biol (2007) 1.16
Susceptibility to cytotoxic T lymphocyte-induced apoptosis is a function of the proliferative status of the target. J Exp Med (1994) 1.12
An extracellular signal-regulated kinase 1- and 2-dependent program of chromatin trafficking of c-Fos and Fra-1 is required for cyclin D1 expression during cell cycle reentry. Mol Cell Biol (2004) 1.12
Sustained activation of mitogen-activated protein kinases and activator protein 1 by the hepatitis B virus X protein in mouse hepatocytes in vivo. J Virol (2001) 1.11
Cellular ras and cyclin D1 are required during different cell cycle periods in cycling NIH 3T3 cells. Mol Cell Biol (1999) 1.11
A C-terminal domain in FosB, absent in FosB/SF and Fra-1, which is able to interact with the TATA binding protein, is required for altered cell growth. EMBO J (1994) 1.08
Elevated phosphate activates N-ras and promotes cell transformation and skin tumorigenesis. Cancer Prev Res (Phila) (2010) 1.07
Transformation by Fos proteins requires a C-terminal transactivation domain. Mol Cell Biol (1993) 1.06
MEK5, a new target of the atypical protein kinase C isoforms in mitogenic signaling. Mol Cell Biol (2001) 1.05
Ligand-specific sequential regulation of transcription factors for differentiation of MCF-7 cells. BMC Genomics (2009) 1.00
Heterodimerization with different Jun proteins controls c-Fos intranuclear dynamics and distribution. J Biol Chem (2010) 0.96
Involvement of JunD in transcriptional activation of the orphan receptor gene nur77 by nerve growth factor and membrane depolarization in PC12 cells. Mol Cell Biol (1994) 0.96
c-Fos activated phospholipid synthesis is required for neurite elongation in differentiating PC12 cells. Mol Biol Cell (2004) 0.95
JunB breakdown in mid-/late G2 is required for down-regulation of cyclin A2 levels and proper mitosis. Mol Cell Biol (2008) 0.93
High-mobility-group protein I can modulate binding of transcription factors to the U5 region of the human immunodeficiency virus type 1 proviral promoter. J Virol (2000) 0.93
PEST motifs are not required for rapid calpain-mediated proteolysis of c-fos protein. Biochem J (1996) 0.91
Examining docking interactions on ERK2 with modular peptide substrates. Biochemistry (2011) 0.86
Characterization of a delayed early serum response region. Mol Cell Biol (1994) 0.86
A T cell controlled molecular pathway regulating the IgH locus: CD40-mediated activation of the IgH 3' enhancer. EMBO J (1996) 0.86
Differential regulation of c-Fos and FosB in the rat brain after amygdala kindling. Cell Mol Neurobiol (2006) 0.83
Anti-apoptotic effects of protein kinase C-delta and c-fos in cisplatin-treated thyroid cells. Br J Pharmacol (2009) 0.83
Tissue-specific transgenic knockdown of Fos-related antigen 2 (Fra-2) expression mediated by dominant negative Fra-2. Mol Cell Biol (2001) 0.83
Sphingosylphosphocholine, a signaling molecule which accumulates in Niemann-Pick disease type A, stimulates DNA-binding activity of the transcription activator protein AP-1. Proc Natl Acad Sci U S A (1995) 0.81
Transformation by the fos or jun oncogene does not increase AP-1 DNA-binding activity. J Virol (1993) 0.80
DT-diaphorase activity in NSCLC and SCLC cell lines: a role for fos/jun regulation. Br J Cancer (1999) 0.80
Identification of a rel-related protein in the nucleus during the S phase of the cell cycle. Mol Cell Biol (1993) 0.80
c-Fos activates and physically interacts with specific enzymes of the pathway of synthesis of polyphosphoinositides. Mol Biol Cell (2011) 0.79
miR-196b Is Epigenetically Silenced during the Premalignant Stage of Lung Carcinogenesis. Cancer Res (2016) 0.77
The Catalytic Efficiency of Lipin 1β Increases by Physically Interacting with the Proto-oncoprotein c-Fos. J Biol Chem (2015) 0.75
Modulation of muscle creatine kinase promoter activity by the inducible orphan nuclear receptor TIS1. Biochem J (1997) 0.75
Stimulation of 3T3 cells induces transcription of the c-fos proto-oncogene. Nature (1984) 24.45
Phorbol ester-inducible genes contain a common cis element recognized by a TPA-modulated trans-acting factor. Cell (1987) 20.61
Purified transcription factor AP-1 interacts with TPA-inducible enhancer elements. Cell (1987) 14.62
The role of Jun, Fos and the AP-1 complex in cell-proliferation and transformation. Biochim Biophys Acta (1991) 11.78
Induction of c-fos gene and protein by growth factors precedes activation of c-myc. Nature (1985) 9.73
Platelet-derived growth factor induces rapid but transient expression of the c-fos gene and protein. Nature (1985) 8.36
A gene activated by growth factors is related to the oncogene v-jun. Proc Natl Acad Sci U S A (1988) 7.47
c-Jun dimerizes with itself and with c-Fos, forming complexes of different DNA binding affinities. Cell (1988) 6.24
DNA binding activities of three murine Jun proteins: stimulation by Fos. Cell (1988) 6.13
c-fos protein can induce cellular transformation: a novel mechanism of activation of a cellular oncogene. Cell (1984) 5.55
Primary response genes induced by growth factors and tumor promoters. Annu Rev Biochem (1991) 5.48
Induction of proto-oncogene JUN/AP-1 by serum and TPA. Nature (1988) 5.18
fra-1: a serum-inducible, cellular immediate-early gene that encodes a fos-related antigen. Mol Cell Biol (1988) 5.12
Transcriptional activation of c-jun during the G0/G1 transition in mouse fibroblasts. Nature (1988) 5.04
The product of a novel growth factor activated gene, fos B, interacts with JUN proteins enhancing their DNA binding activity. EMBO J (1989) 4.27
Jun-B differs in its biological properties from, and is a negative regulator of, c-Jun. Cell (1989) 4.17
Inducible production of c-fos antisense RNA inhibits 3T3 cell proliferation. Proc Natl Acad Sci U S A (1986) 3.71
Fos and Jun bind cooperatively to the AP-1 site: reconstitution in vitro. Genes Dev (1988) 3.70
jun-D: a third member of the jun gene family. Proc Natl Acad Sci U S A (1989) 3.47
c-JUN, JUN B, and JUN D differ in their binding affinities to AP-1 and CRE consensus sequences: effect of FOS proteins. Oncogene (1991) 3.41
A naturally occurring truncated form of FosB that inhibits Fos/Jun transcriptional activity. Cell (1991) 3.12
The jun and fos protein families are both required for cell cycle progression in fibroblasts. Mol Cell Biol (1991) 3.08
jun-B inhibits and c-fos stimulates the transforming and trans-activating activities of c-jun. Cell (1989) 3.05
Antisense RNA of proto-oncogene c-fos blocks renewed growth of quiescent 3T3 cells. Mol Cell Biol (1987) 2.97
Induction of protooncogene c-jun by serum growth factors. Proc Natl Acad Sci U S A (1988) 2.92
The product of a fos-related gene, fra-1, binds cooperatively to the AP-1 site with Jun: transcription factor AP-1 is comprised of multiple protein complexes. Genes Dev (1989) 2.41
Isolation and characterization of fra-2, an additional member of the fos gene family. Proc Natl Acad Sci U S A (1990) 2.40
Isolation of human fos-related genes and their expression during monocyte-macrophage differentiation. Oncogene (1990) 2.17
Deregulated expression of human c-jun transforms primary rat embryo cells in cooperation with an activated c-Ha-ras gene and transforms rat-1a cells as a single gene. Proc Natl Acad Sci U S A (1989) 2.13
Microinjection of fos-specific antibodies blocks DNA synthesis in fibroblast cells. Mol Cell Biol (1988) 2.00
Expression of different Jun and Fos proteins during the G0-to-G1 transition in mouse fibroblasts: in vitro and in vivo associations. Mol Cell Biol (1991) 1.91
Activation of the transforming potential of the human fos proto-oncogene requires message stabilization and results in increased amounts of partially modified fos protein. Mol Cell Biol (1988) 1.70
An alternative spliced form of FosB is a negative regulator of transcriptional activation and transformation by Fos proteins. Proc Natl Acad Sci U S A (1991) 1.54
Difference in transcriptional regulatory function between c-Fos and Fra-2. Nucleic Acids Res (1991) 1.43
Single cell assay with an automated capillary microinjection system. Exp Cell Res (1988) 1.43
Both products of the fosB gene, FosB and its short form, FosB/SF, are transcriptional activators in fibroblasts. Mol Cell Biol (1991) 1.23
Inhibition of c-jun causes reversible proliferative arrest and withdrawal from the cell cycle. Blood (1992) 1.08
Constitutive expression of FosB and its short form, FosB/SF, induces malignant cell transformation in rat-1A cells. New Biol (1991) 0.97
fosB is a transforming gene encoding a transcriptional activator. Oncogene (1991) 0.90
Induction of c-fos gene and protein by growth factors precedes activation of c-myc. Nature (1985) 9.73
Cyclin/PCNA is the auxiliary protein of DNA polymerase-delta. Nature (1987) 8.28
Osteopetrosis in mice lacking NF-kappaB1 and NF-kappaB2. Nat Med (1997) 5.72
Complexity of the early genetic response to growth factors in mouse fibroblasts. Mol Cell Biol (1988) 5.21
Transcriptional activation of c-jun during the G0/G1 transition in mouse fibroblasts. Nature (1988) 5.04
Multiorgan inflammation and hematopoietic abnormalities in mice with a targeted disruption of RelB, a member of the NF-kappa B/Rel family. Cell (1995) 4.70
Defects in macrophage recruitment and host defense in mice lacking the CCR2 chemokine receptor. J Exp Med (1997) 4.57
The product of a novel growth factor activated gene, fos B, interacts with JUN proteins enhancing their DNA binding activity. EMBO J (1989) 4.27
Two mouse genes encoding potential transcription factors with identical DNA-binding domains are activated by growth factors in cultured cells. Proc Natl Acad Sci U S A (1988) 4.08
RelB, a new Rel family transcription activator that can interact with p50-NF-kappa B. Mol Cell Biol (1992) 4.03
A gene encoding a protein with zinc fingers is activated during G0/G1 transition in cultured cells. EMBO J (1988) 3.98
A novel mitogen-inducible gene product related to p50/p105-NF-kappa B participates in transactivation through a kappa B site. Mol Cell Biol (1992) 3.63
Characterization of junD: a new member of the jun proto-oncogene family. EMBO J (1989) 3.55
A search for differential polypeptide synthesis throughout the cell cycle of HeLa cells. J Cell Biol (1980) 3.50
c-JUN, JUN B, and JUN D differ in their binding affinities to AP-1 and CRE consensus sequences: effect of FOS proteins. Oncogene (1991) 3.41
Activation of the nuclear factor-kappaB by Rho, CDC42, and Rac-1 proteins. Genes Dev (1997) 3.23
The jun and fos protein families are both required for cell cycle progression in fibroblasts. Mol Cell Biol (1991) 3.08
Cloning and sequence of the human nuclear protein cyclin: homology with DNA-binding proteins. Proc Natl Acad Sci U S A (1987) 3.05
The leucine repeat motif in Fos protein mediates complex formation with Jun/AP-1 and is required for transformation. Cell (1989) 2.84
An enzymatic assay reveals that proteins destined for the apical or basolateral domains of an epithelial cell line share the same late Golgi compartments. EMBO J (1985) 2.78
Nuclear factor (NF)-kappa B2 (p100/p52) is required for normal splenic microarchitecture and B cell-mediated immune responses. J Exp Med (1998) 2.78
Altered gene expression in neurons during programmed cell death: identification of c-jun as necessary for neuronal apoptosis. J Cell Biol (1994) 2.75
Controlled recruitment of monocytes and macrophages to specific organs through transgenic expression of monocyte chemoattractant protein-1. J Immunol (1995) 2.47
Changes in the nuclear distribution of cyclin (PCNA) but not its synthesis depend on DNA replication. EMBO J (1985) 2.28
Rapid induction in regenerating liver of RL/IF-1 (an I kappa B that inhibits NF-kappa B, RelB-p50, and c-Rel-p50) and PHF, a novel kappa B site-binding complex. Mol Cell Biol (1992) 2.27
Disruption of the erp/mkp-1 gene does not affect mouse development: normal MAP kinase activity in ERP/MKP-1-deficient fibroblasts. Oncogene (1996) 2.20
Segment-specific expression of a zinc-finger gene in the developing nervous system of the mouse. Nature (1989) 2.18
Gastric hyperplasia and increased proliferative responses of lymphocytes in mice lacking the COOH-terminal ankyrin domain of NF-kappaB2. J Exp Med (1997) 2.13
Architecture and polypeptide composition of HeLa cytoskeletons. Modification of cytoarchitectural polypeptides during mitosis. J Mol Biol (1982) 2.13
The presence of dialkylphosphates in fresh fruit juices: implication for organophosphorus pesticide exposure and risk assessments. J Toxicol Environ Health A (2005) 2.05
Characterization of a mouse multigene family that encodes zinc finger structures. Mol Cell Biol (1988) 2.05
Identification of a nuclear and of a cytoplasmic polypeptide whose relative proportions are sensitive to changes in the rate of cell proliferation. Exp Cell Res (1981) 1.99
Structure, mapping and expression of a growth factor inducible gene encoding a putative nuclear hormonal binding receptor. EMBO J (1989) 1.98
Cloning and functional expression of mCCR2, a murine receptor for the C-C chemokines JE and FIC. J Biol Chem (1996) 1.94
The NFAT-1 DNA binding complex in activated T cells contains Fra-1 and JunB. Mol Cell Biol (1993) 1.94
Synthesis of the nuclear protein cyclin (PCNA) and its relationship with DNA replication. Exp Cell Res (1986) 1.92
Impaired macrophage function and enhanced T cell-dependent immune response in mice lacking CCR5, the mouse homologue of the major HIV-1 coreceptor. J Immunol (1998) 1.92
Expression of different Jun and Fos proteins during the G0-to-G1 transition in mouse fibroblasts: in vitro and in vivo associations. Mol Cell Biol (1991) 1.91
Involvement of common and cell type-specific pathways in c-fos gene control: stable induction of cAMP in macrophages. Cell (1987) 1.83
The immediate-early growth response in regenerating liver and insulin-stimulated H-35 cells: comparison with serum-stimulated 3T3 cells and identification of 41 novel immediate-early genes. Mol Cell Biol (1991) 1.82
Structure, mapping, and expression of fisp-12, a growth factor-inducible gene encoding a secreted cysteine-rich protein. Cell Growth Differ (1991) 1.80
Chronic inflammation and susceptibility to bacterial infections in mice lacking the polypeptide (p)105 precursor (NF-kappaB1) but expressing p50. J Exp Med (1998) 1.79
Changes in cyclin/proliferating cell nuclear antigen distribution during DNA repair synthesis. J Cell Biol (1988) 1.79
Cyclin: a nuclear protein whose level correlates directly with the proliferative state of normal as well as transformed cells. Leuk Res (1984) 1.79
Structure, chromosome location, and expression of the mouse zinc finger gene Krox-20: multiple gene products and coregulation with the proto-oncogene c-fos. Mol Cell Biol (1989) 1.78
Exocytotic pathways exist to both the apical and the basolateral cell surface of the polarized epithelial cell MDCK. Cell (1985) 1.76
Stimulation and inhibition of growth by EGF in different A431 cell clones is accompanied by the rapid induction of c-fos and c-myc proto-oncogenes. EMBO J (1985) 1.73
Up-dated catalogue of HeLa cell proteins: percentages and characteristics of the major cell polypeptides labeled with a mixture of 16 14C-labeled amino acids. Clin Chem (1982) 1.70
Phosphorylation changes associated with the early cell cycle in Xenopus eggs. Dev Biol (1987) 1.65
Interactions among LRF-1, JunB, c-Jun, and c-Fos define a regulatory program in the G1 phase of liver regeneration. Mol Cell Biol (1992) 1.64
Structure, mapping, and expression of erp, a growth factor-inducible gene encoding a nontransmembrane protein tyrosine phosphatase, and effect of ERP on cell growth. Mol Cell Biol (1993) 1.62
Constitutive and inducible Rel/NF-kappa B activities in mouse thymus and spleen. Oncogene (1994) 1.59
Human proteins sensitive to neoplastic transformation in cultured epithelial and fibroblast cells. Clin Chem (1982) 1.57
Putative association of mitochondria with a subpopulation of intermediate-sized filaments in cultured human skin fibroblasts. Cell (1982) 1.54
Regulation of c-fos transcription in mouse fibroblasts: identification of DNase I-hypersensitive sites and regulatory upstream sequences. EMBO J (1985) 1.51
Expression of relB transcripts during lymphoid organ development: specific expression in dendritic antigen-presenting cells. Development (1993) 1.48
Both N- and C-terminal domains of RelB are required for full transactivation: role of the N-terminal leucine zipper-like motif. Mol Cell Biol (1993) 1.46
Functional redundancy of the nuclear factor kappa B inhibitors I kappa B alpha and I kappa B beta. J Exp Med (1998) 1.45
c-Fos-induced activation of a TATA-box-containing promoter involves direct contact with TATA-box-binding protein. Mol Cell Biol (1994) 1.42
Wounding a fibroblast monolayer results in the rapid induction of the c-fos proto-oncogene. EMBO J (1986) 1.42
Expression of c-fos in NIH3T3 cells is very low but inducible throughout the cell cycle. EMBO J (1986) 1.41
B cells genetically deficient in the c-Rel transactivation domain have selective defects in germline CH transcription and Ig class switching. J Immunol (1997) 1.38
[Interrelationship between the duration of HIV infection, viral load and CD4 positive lymphocyte count]. Med Clin (Barc) (1998) 1.38
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Induction of the nuclear protein 'cyclin' in quiescent mouse 3T3 cells stimulated by serum and growth factors. Correlation with DNA synthesis. EMBO J (1984) 1.36
Multifocal defects in immune responses in RelB-deficient mice. J Immunol (1997) 1.36
[35S]-methionine labelled polypeptides from HELA cells. Coordinates and percentage of some major polypeptides. Cell Biol Int Rep (1981) 1.34
Role of nuclear material in the early cell cycle of Xenopus embryos. Cell (1988) 1.34
B cells lacking RelB are defective in proliferative responses, but undergo normal B cell maturation to Ig secretion and Ig class switching. J Exp Med (1996) 1.34
Developmental expression of the mouse c-rel proto-oncogene in hematopoietic organs. Development (1994) 1.34
Expression of the chemokine N51/KC in the thymus and epidermis of transgenic mice results in marked infiltration of a single class of inflammatory cells. J Exp Med (1994) 1.33
Antigen-induced eosinophilic lung inflammation develops in mice deficient in chemokine eotaxin. Blood (1998) 1.33
p50-NF-kappaB complexes partially compensate for the absence of RelB: severely increased pathology in p50(-/-)relB(-/-) double-knockout mice. J Exp Med (1997) 1.32
Tissue-specific expression of c-jun and junB during organogenesis in the mouse. Development (1989) 1.29
Activation of the granulocyte-macrophage colony-stimulating factor promoter in T cells requires cooperative binding of Elf-1 and AP-1 transcription factors. Mol Cell Biol (1994) 1.28
Coexistence of three major isoactins in a single sarcoma 180 cell. Cell (1981) 1.28
Structure and mapping of the fosB gene. FosB downregulates the activity of the fosB promoter. Nucleic Acids Res (1992) 1.24
Constitutive expression of Bc1-3 in thymocytes increases the DNA binding of NF-kappaB1 (p50) homodimers in vivo. Mol Cell Biol (1996) 1.24
Gene expression in normal and virally transformed mouse 3T3b and hamster BHK21 cells. Exp Cell Res (1980) 1.24
Differential interactions of Rel-NF-kappa B complexes with I kappa B alpha determine pools of constitutive and inducible NF-kappa B activity. EMBO J (1994) 1.23
Both products of the fosB gene, FosB and its short form, FosB/SF, are transcriptional activators in fibroblasts. Mol Cell Biol (1991) 1.23
Both multiorgan inflammation and myeloid hyperplasia in RelB-deficient mice are T cell dependent. J Immunol (1996) 1.23
The NF-kappa B family member RelB is required for innate and adaptive immunity to Toxoplasma gondii. J Immunol (1999) 1.22
Specific inhibition of RelB/p52 transcriptional activity by the C-terminal domain of p100. Oncogene (1995) 1.22
Apoptosis of nur77/N10-transgenic thymocytes involves the Fas/Fas ligand pathway. Proc Natl Acad Sci U S A (1996) 1.22
Epidermal growth factor inhibits the synthesis of the nuclear protein cyclin in A431 human carcinoma cells. Proc Natl Acad Sci U S A (1984) 1.22
[35S]-methionine labelled polypeptides from secondary mouse kidney fibroblasts: coordinates and one dimensional peptide maps of some major polypeptides. Cell Biol Int Rep (1981) 1.21
Coordinated synthesis of the nuclear protein cyclin and DNA in serum-stimulated quiescent 3T3 cells. FEBS Lett (1984) 1.20
Bombesin induces c-fos and c-myc expression in quiescent Swiss 3T3 cells. Comparative study with other mitogens. Exp Cell Res (1987) 1.19
Gene expression in murine hybrids exhibiting different morphologies and tumorigenic properties. Carcinogenesis (1981) 1.18
Overexpression of RelA in transgenic mouse thymocytes: specific increase in levels of the inhibitor protein I kappa B alpha. Mol Cell Biol (1995) 1.16
Regulation of Fra-1 and Fra-2 phosphorylation differs during the cell cycle of fibroblasts and phosphorylation in vitro by MAP kinase affects DNA binding activity. Oncogene (1994) 1.15
Synthesis of the nuclear protein cyclin in growing, senescent and morphologically transformed human skin fibroblasts. FEBS Lett (1984) 1.15
Neutrophil infiltration, glial reaction, and neurological disease in transgenic mice expressing the chemokine N51/KC in oligodendrocytes. J Clin Invest (1996) 1.13
Idiopathic CD4+ T-lymphocytopenia. Lancet (1992) 1.12
NOT, a human immediate-early response gene closely related to the steroid/thyroid hormone receptor NAK1/TR3. Mol Endocrinol (1994) 1.12
Genetic approaches to study Rel/NF-kappa B/I kappa B function in mice. Semin Cancer Biol (1997) 1.12
The v-rel oncogene promotes malignant T-cell leukemia/lymphoma in transgenic mice. EMBO J (1996) 1.12