Published in J Leukoc Biol on February 04, 2005
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IL-32, a proinflammatory cytokine in rheumatoid arthritis. Proc Natl Acad Sci U S A (2006) 2.67
Silencing of Fas, but not caspase-8, in lung epithelial cells ameliorates pulmonary apoptosis, inflammation, and neutrophil influx after hemorrhagic shock and sepsis. Am J Pathol (2005) 1.60
Lung injury after hemorrhage is age dependent: role of peroxisome proliferator-activated receptor gamma. Crit Care Med (2009) 1.55
Therapeutic accessibility of caspase-mediated cell death as a key pathomechanism in indirect acute lung injury. Crit Care Med (2010) 1.49
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Intrapulmonary delivery of XCL1-targeting small interfering RNA in mice chronically infected with Mycobacterium tuberculosis. Am J Respir Cell Mol Biol (2008) 0.92
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Silencing airway epithelial cell-derived hepcidin exacerbates sepsis induced acute lung injury. Crit Care (2014) 0.82
The role and source of tumor necrosis factor-α in hemorrhage-induced priming for septic lung injury. Shock (2012) 0.82
In silico identification and expression analysis of 12 novel CC chemokines in catfish. Immunogenetics (2005) 0.81
Local tissue expression of the cell death ligand, fas ligand, plays a central role in the development of extrapulmonary acute lung injury. Shock (2011) 0.81
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RNA interference as a potential therapeutic treatment for inflammation associated lung injury. Int J Clin Exp Med (2008) 0.79
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Elastase induces lung epithelial cell autophagy through placental growth factor: a new insight of emphysema pathogenesis. Autophagy (2014) 0.78
PlGF mediates neutrophil elastase-induced airway epithelial cell apoptosis and emphysema. Respir Res (2014) 0.77
Stimulation of Brain AMP-Activated Protein Kinase Attenuates Inflammation and Acute Lung Injury in Sepsis. Mol Med (2015) 0.77
Post-transcriptional silencing of CCR3 downregulates IL-4 stimulated release of eotaxin-3 (CCL26) and other CCR3 ligands in alveolar type II cells. Cytokine (2008) 0.76
Immunological research using RNA interference technology. Immunology (2007) 0.75
Platelet Activating Factor Receptor Activation Improves siRNA Uptake and RNAi Responses in Well-differentiated Airway Epithelia. Mol Ther Nucleic Acids (2014) 0.75
MyD88-dependent expansion of an immature GR-1(+)CD11b(+) population induces T cell suppression and Th2 polarization in sepsis. J Exp Med (2007) 5.48
PD-1 expression by macrophages plays a pathologic role in altering microbial clearance and the innate inflammatory response to sepsis. Proc Natl Acad Sci U S A (2009) 4.23
Leukocyte apoptosis and its significance in sepsis and shock. J Leukoc Biol (2005) 2.38
Increased circulating regulatory T cells (CD4(+)CD25 (+)CD127 (-)) contribute to lymphocyte anergy in septic shock patients. Intensive Care Med (2008) 2.34
In vivo delivery of caspase-8 or Fas siRNA improves the survival of septic mice. Blood (2005) 1.99
Regulatory T cell populations in sepsis and trauma. J Leukoc Biol (2007) 1.96
The compensatory anti-inflammatory response syndrome (CARS) in critically ill patients. Clin Chest Med (2008) 1.86
Shock-induced neutrophil mediated priming for acute lung injury in mice: divergent effects of TLR-4 and TLR-4/FasL deficiency. Am J Pathol (2002) 1.78
Cardiopulmonary, histological, and inflammatory alterations after lung contusion in a novel mouse model of blunt chest trauma. Shock (2003) 1.78
Programmed death-1 levels correlate with increased mortality, nosocomial infection and immune dysfunctions in septic shock patients. Crit Care (2011) 1.78
The contribution of CD4+ CD25+ T-regulatory-cells to immune suppression in sepsis. Shock (2007) 1.71
Pulmonary contusion causes impairment of macrophage and lymphocyte immune functions and increases mortality associated with a subsequent septic challenge. Crit Care Med (2005) 1.65
Silencing of Fas, but not caspase-8, in lung epithelial cells ameliorates pulmonary apoptosis, inflammation, and neutrophil influx after hemorrhagic shock and sepsis. Am J Pathol (2005) 1.60
Changes and regulation of the C5a receptor on neutrophils during septic shock in humans. J Immunol (2013) 1.57
Tumor necrosis factor-alpha-stimulated polymorphonuclear leukocytes suppress migration and bactericidal activity of polymorphonuclear leukocytes in a paracrine manner. Crit Care Med (2002) 1.52
Therapeutic accessibility of caspase-mediated cell death as a key pathomechanism in indirect acute lung injury. Crit Care Med (2010) 1.49
The apoptotic pathway as a therapeutic target in sepsis. Curr Drug Targets (2007) 1.47
Fas-induced pulmonary apoptosis and inflammation during indirect acute lung injury. Am J Respir Crit Care Med (2007) 1.47
Role of alveolar macrophage and migrating neutrophils in hemorrhage-induced priming for ALI subsequent to septic challenge. Am J Physiol Lung Cell Mol Physiol (2005) 1.40
Inhibition of Fas/Fas ligand signaling improves septic survival: differential effects on macrophage apoptotic and functional capacity. J Leukoc Biol (2003) 1.39
Female sex hormones regulate macrophage function after trauma-hemorrhage and prevent increased death rate from subsequent sepsis. Ann Surg (2002) 1.39
CXCR2 inhibition suppresses hemorrhage-induced priming for acute lung injury in mice. J Leukoc Biol (2004) 1.34
Treatment with GITR agonistic antibody corrects adaptive immune dysfunction in sepsis. Blood (2007) 1.29
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Failure to normalize lymphopenia following trauma is associated with increased mortality, independent of the leukocytosis pattern. Crit Care (2012) 1.23
BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction. J Leukoc Biol (2012) 1.22
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Polymicrobial sepsis induces divergent effects on splenic and peritoneal dendritic cell function in mice. Shock (2004) 1.22
Decreased monocyte human leukocyte antigen-DR expression after severe burn injury: Correlation with severity and secondary septic shock. Crit Care Med (2007) 1.18
Pathogenesis of indirect (secondary) acute lung injury. Expert Rev Respir Med (2011) 1.16
CD8+ T cells promote inflammation and apoptosis in the liver after sepsis: role of Fas-FasL. Am J Pathol (2007) 1.15
The role of C5a in the innate immune response after experimental blunt chest trauma. Shock (2008) 1.14
CD4+ lymphocytes control gut epithelial apoptosis and mediate survival in sepsis. FASEB J (2009) 1.14
Differential alterations in cardiovascular responses during the progression of polymicrobial sepsis in the mouse. Shock (2002) 1.11
Recombinant human activated protein C inhibits integrin-mediated neutrophil migration. Blood (2009) 1.11
Macrophage arginase regulation by CCAAT/enhancer-binding protein beta. Shock (2005) 1.11
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Lymphocytes in the development of lung inflammation: a role for regulatory CD4+ T cells in indirect pulmonary lung injury. J Immunol (2009) 1.07
Role of activated neutrophils in chest trauma-induced septic acute lung injury. Shock (2012) 1.05
The role of hepatic invariant NKT cells in systemic/local inflammation and mortality during polymicrobial septic shock. J Immunol (2009) 1.04
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B and T lymphocyte attenuator expression on CD4+ T-cells associates with sepsis and subsequent infections in ICU patients. Crit Care (2013) 1.03
Sepsis-induced changes in macrophage co-stimulatory molecule expression: CD86 as a regulator of anti-inflammatory IL-10 response. Surg Infect (Larchmt) (2004) 1.01
Deficiency of gammadelta T lymphocytes contributes to mortality and immunosuppression in sepsis. Am J Physiol Regul Integr Comp Physiol (2006) 1.01
Programmed death 1 expression as a marker for immune and physiological dysfunction in the critically ill surgical patient. Shock (2012) 0.99
The dual functions of receptor interacting protein 1 in fas-induced hepatocyte death during sepsis. Shock (2011) 0.97
Early complementopathy after multiple injuries in humans. Shock (2012) 0.97
Plasmacytoid dendritic cells control lung inflammation and monocyte recruitment in indirect acute lung injury in mice. Am J Pathol (2009) 0.97
Mechanisms of indirect acute lung injury: a novel role for the coinhibitory receptor, programmed death-1. Ann Surg (2012) 0.97
Pulmonary contusion induces alveolar type 2 epithelial cell apoptosis: role of alveolar macrophages and neutrophils. Shock (2008) 0.96
Genetic disruption of protein kinase Cδ reduces endotoxin-induced lung injury. Am J Physiol Lung Cell Mol Physiol (2012) 0.95
Biochemical characterization of individual injury pattern and injury severity. Injury (2003) 0.94
Divergent roles of murine neutrophil chemokines in hemorrhage induced priming for acute lung injury. Cytokine (2005) 0.94
Contribution of anti-inflammatory/immune suppressive processes to the pathology of sepsis. Front Biosci (2006) 0.93
Inhibition of CD1d activation suppresses septic mortality: a role for NK-T cells in septic immune dysfunction. J Surg Res (2003) 0.93
Pathological aspects of apoptosis in severe sepsis and shock? Int J Biochem Cell Biol (2003) 0.93
Sex-specific p38 MAP kinase activation following trauma-hemorrhage: involvement of testosterone and estradiol. Am J Physiol Endocrinol Metab (2003) 0.91
Inflammatory alterations in a novel combination model of blunt chest trauma and hemorrhagic shock. J Trauma (2011) 0.90
Deficiency of Bid protein reduces sepsis-induced apoptosis and inflammation, while improving septic survival. Shock (2010) 0.90
Liver myeloid-derived suppressor cells expand in response to liver metastases in mice and inhibit the anti-tumor efficacy of anti-CEA CAR-T. Cancer Immunol Immunother (2015) 0.90
SOCS-1 is a central mediator of steroid-increased thymocyte apoptosis and decreased survival following sepsis. Apoptosis (2007) 0.89
Activation of common antiviral pathways can potentiate inflammatory responses to septic shock. Shock (2006) 0.88
Kupffer cells protect liver sinusoidal endothelial cells from Fas-dependent apoptosis in sepsis by down-regulating gp130. Am J Pathol (2013) 0.88