Published in Shock on May 01, 2011
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CD8+ T cells promote inflammation and apoptosis in the liver after sepsis: role of Fas-FasL. Am J Pathol (2007) 1.15
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Recombinant human activated protein C inhibits integrin-mediated neutrophil migration. Blood (2009) 1.11
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Lymphocytes in the development of lung inflammation: a role for regulatory CD4+ T cells in indirect pulmonary lung injury. J Immunol (2009) 1.07
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Deficiency of gammadelta T lymphocytes contributes to mortality and immunosuppression in sepsis. Am J Physiol Regul Integr Comp Physiol (2006) 1.01
Programmed death 1 expression as a marker for immune and physiological dysfunction in the critically ill surgical patient. Shock (2012) 0.99
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Inhibition of CD1d activation suppresses septic mortality: a role for NK-T cells in septic immune dysfunction. J Surg Res (2003) 0.93
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Sex-specific p38 MAP kinase activation following trauma-hemorrhage: involvement of testosterone and estradiol. Am J Physiol Endocrinol Metab (2003) 0.91
Deficiency of Bid protein reduces sepsis-induced apoptosis and inflammation, while improving septic survival. Shock (2010) 0.90
Liver myeloid-derived suppressor cells expand in response to liver metastases in mice and inhibit the anti-tumor efficacy of anti-CEA CAR-T. Cancer Immunol Immunother (2015) 0.90
SOCS-1 is a central mediator of steroid-increased thymocyte apoptosis and decreased survival following sepsis. Apoptosis (2007) 0.89
Kupffer cells protect liver sinusoidal endothelial cells from Fas-dependent apoptosis in sepsis by down-regulating gp130. Am J Pathol (2013) 0.88
Activation of common antiviral pathways can potentiate inflammatory responses to septic shock. Shock (2006) 0.88
Apoptosis. Crit Care Med (2005) 0.88
Blockade of apoptosis as a rational therapeutic strategy for the treatment of sepsis. Novartis Found Symp (2007) 0.86
Identification of B7-H1 as a novel mediator of the innate immune/proinflammatory response as well as a possible myeloid cell prognostic biomarker in sepsis. J Immunol (2013) 0.86
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Paracrine suppression of apoptosis by cytokine-stimulated neutrophils involves divergent regulation of NF-kappaB, Bcl-X(L), and Bak. Shock (2002) 0.86
Beneficial versus detrimental effects of neutrophils are determined by the nature of the insult. J Am Coll Surg (2007) 0.86
NF-kappaB activation has tissue-specific effects on immune cell apoptosis during polymicrobial sepsis. Shock (2002) 0.85
Kupffer cells potentiate liver sinusoidal endothelial cell injury in sepsis by ligating programmed cell death ligand-1. J Leukoc Biol (2013) 0.85
Polymicrobial sepsis enhances clearance of apoptotic immune cells by splenic macrophages. Surgery (2007) 0.85