Glucose substitution prolongs maintenance of energy homeostasis and lifespan of telomere dysfunctional mice.

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🔗 View Article (PMC 4199114)

Published in Nat Commun on September 18, 2014

Authors

Pavlos Missios1, Yuan Zhou1, Luis Miguel Guachalla1, Guido von Figura1, Andre Wegner2, Sundaram Reddy Chakkarappan1, Tina Binz2, Anne Gompf3, Götz Hartleben3, Martin D Burkhalter3, Veronika Wulff4, Cagatay Günes3, Rui Wang Sattler5, Zhangfa Song1, Thomas Illig5, Susanne Klaus6, Bernhard O Böhm7, Tina Wenz4, Karsten Hiller2, K Lenhard Rudolph3

Author Affiliations

1: Cooperation Group of the Leibniz Institute for Age Research-Fritz-Lipmann-Institute (FLI) Jena with the University of Ulm, 89081 Ulm, Germany.
2: Luxembourg Centre for Systems Biomedicine, University of Luxembourg, 7, avenue des Hauts-Fourneaux, Esch-Belval L-4362, Luxembourg.
3: Leibniz Institute for Age Research-Fritz Lipmann Institute (FLI), Beutenbergstr 11, 07745 Jena, Germany.
4: Institute for Genetics, Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases (CECAD), University of Cologne, Zülpicher Street 47A, 50674 Cologne, Germany.
5: Institute of Epidemiology, Ingolstädter Landstrasse 1, 85764 Munich/Neuherberg, Germany.
6: German Institute of Human Nutrition, Arthur-Scheunert-Allee 114-116, 14558 Nuthetal, Germany.
7: Department of Internal Medicine I, University of Ulm, 89081 Ulm, Germany.

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