Neutrophil extracellular trap (NET) formation characterises stable and exacerbated COPD and correlates with airflow limitation.

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Published in Respir Res on May 22, 2015

Authors

Fikreta Grabcanovic-Musija1, Astrid Obermayer2, Walter Stoiber3, Wolf-Dietrich Krautgartner4, Peter Steinbacher5, Nicole Winterberg6, Arne Cornelius Bathke7, Michaela Klappacher8, Michael Studnicka9

Author Affiliations

1: University Clinic of Pneumology, Paracelsus Medical University, Müllner Hauptstraße 48, A-5020, Salzburg, Austria. f.grabcanovic-musija@salk.at.
2: Department of Cell Biology, Biomedical Ultrastructure Research Group, University of Salzburg, Salzburg, Austria. astrid.obermayer@sbg.ac.at.
3: Department of Cell Biology, Biomedical Ultrastructure Research Group, University of Salzburg, Salzburg, Austria. walter.stoiber@sbg.ac.at.
4: Department of Cell Biology, Biomedical Ultrastructure Research Group, University of Salzburg, Salzburg, Austria. wolf.krautgartner@sbg.ac.at.
5: Department of Cell Biology, Biomedical Ultrastructure Research Group, University of Salzburg, Salzburg, Austria. peter.steinbacher@sbg.ac.at.
6: Department of Mathematics, University of Salzburg, Salzburg, Austria. nicole.winterberg@sbg.ac.at.
7: Department of Mathematics, University of Salzburg, Salzburg, Austria. arne.bathke@sbg.ac.at.
8: Department of Cell Biology, Biomedical Ultrastructure Research Group, University of Salzburg, Salzburg, Austria. michaela.klappacher@sbg.ac.at.
9: University Clinic of Pneumology, Paracelsus Medical University, Müllner Hauptstraße 48, A-5020, Salzburg, Austria. m.studnicka@salk.at.

Associated clinical trials:

Sputum Studies of Anti-Citrullinated Protein Antibodies (ACPA) and Rheumatoid Arthritis (RA) Origins (SPARO) | NCT04474392

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