Increased human AP endonuclease 1 level confers protection against the paternal age effect in mice.

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Published in Mutat Res on June 20, 2015

Authors

Jamila R Sanchez1, Traci L Reddick2, Marissa Perez1, Victoria E Centonze3, Sankar Mitra4, Tadahide Izumi5, C Alex McMahan6, Christi A Walter7

Author Affiliations

1: Department of Cellular and Structural Biology, The University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Dr., San Antonio, TX 78229, USA.
2: Duke Cancer Institute, Duke University, 20 Duke Medicine Circle, Durham, NC 27710, USA.
3: Cell & Tissue Imaging Center, St. Jude Children's Research Hospital, 262 Danny Thomas Place, Memphis, TN 38105, USA.
4: Department of Radiation Oncology, Houston Methodist Research Institute, 6550 Fannin Street, Houston, TX 77030, USA.
5: Graduate Center for Toxicology, 1095 V.A. Drive, Lexington, KY 40536, USA.
6: Department of Pathology, The University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Dr., San Antonio, TX 78229, USA.
7: South Texas Veteran's Health Care System, 7400 Merton Minter Blvd., San Antonio, TX 78229, USA; Department of Cellular and Structural Biology, The University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Dr., San Antonio, TX 78229, USA; Cancer Therapy & Research Center, The University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Dr., San Antonio, TX 78229, USA; Barshop Institute for Longevity and Aging Studies, The University of Texas Health Science Center at San Antonio, 7703 Floyd Curl Dr., San Antonio, TX 78229, USA. Electronic address: walter@uthscsa.edu.

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