Tissue-Resident NK Cells Mediate Ischemic Kidney Injury and Are Not Depleted by Anti-Asialo-GM1 Antibody.

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Published in J Immunol on October 09, 2015

Authors

Francisco Victorino1, Dorothy K Sojka2, Kelley S Brodsky3, Eoin N McNamee3, Joanne C Masterson4, Dirk Homann3, Wayne M Yokoyama5, Holger K Eltzschig3, Eric T Clambey6

Author Affiliations

1: Department of Anesthesiology, University of Colorado School of Medicine, Aurora, CO 80045; Immunology Graduate Program, University of Colorado School of Medicine, Aurora, CO 80045;
2: Rheumatology Division, Washington University School of Medicine, St. Louis, MO 63110;
3: Department of Anesthesiology, University of Colorado School of Medicine, Aurora, CO 80045;
4: Gastrointestinal Eosinophilic Diseases Program, Section of Pediatric Gastroenterology, Hepatology and Nutrition, Department of Pediatrics, University of Colorado School of Medicine, Aurora, CO 80045; Digestive Health Institute, Children's Hospital Colorado, Aurora, CO 80045; and.
5: Rheumatology Division, Washington University School of Medicine, St. Louis, MO 63110; Howard Hughes Medical Institute, Washington University School of Medicine, St. Louis, MO 63110.
6: Department of Anesthesiology, University of Colorado School of Medicine, Aurora, CO 80045; Eric.Clambey@ucdenver.edu.

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