Published in J Neurosci on August 01, 2000
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The role of an inwardly rectifying K(+) channel (Kir4.1) in the inner ear and hearing loss. Neuroscience (2014) 0.94
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Immunocytochemical studies of aquaporin 4, Kir4.1, and α1-syntrophin in the astrocyte endfeet of mouse brain capillaries. Acta Histochem Cytochem (2010) 0.87
Inwardly rectifying potassium channel Kir4.1 is responsible for the native inward potassium conductance of satellite glial cells in sensory ganglia. Neuroscience (2010) 0.87
Vascular inward rectifier K+ channels as external K+ sensors in the control of cerebral blood flow. Microcirculation (2015) 0.86
The Membrane Properties of Cochlear Root Cells are Consistent with Roles in Potassium Recirculation and Spatial Buffering. J Assoc Res Otolaryngol (2010) 0.86
Astrocyte regulation of blood flow in the brain. Cold Spring Harb Perspect Biol (2015) 0.86
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Probing potassium channel function in vivo by intracellular delivery of antibodies in a rat model of retinal neurodegeneration. Proc Natl Acad Sci U S A (2010) 0.86
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Glial and neuronal dysfunction in streptozotocin-induced diabetic rats. J Ocul Biol Dis Infor (2011) 0.82
Founder mutation in KCNJ10 in Pakistani patients with EAST syndrome. Mol Genet Genomic Med (2016) 0.82
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Role of astrocytes in epilepsy. Cold Spring Harb Perspect Med (2015) 0.81
Expression of Kir4.1 and Kir5.1 inwardly rectifying potassium channels in oligodendrocytes, the myelinating cells of the CNS. Brain Struct Funct (2016) 0.80
Quantitative trait locus on distal chromosome 1 regulates the occurrence of spontaneous spike-wave discharges in DBA/2 mice. Epilepsia (2012) 0.80
Disruption of KCNJ10 (Kir4.1) stimulates the expression of ENaC in the collecting duct. Am J Physiol Renal Physiol (2016) 0.80
The effect of intraocular and intracranial pressure on retinal structure and function in rats. Physiol Rep (2015) 0.80
Vasopressin-induced stimulation of the Na(+)-activated K(+) channels is responsible for maintaining the basolateral K(+) conductance of the thick ascending limb (TAL) in EAST/SeSAME syndrome. Biochim Biophys Acta (2015) 0.80
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