Transient estrogen receptor binding and p300 redistribution support a squelching mechanism for estradiol-repressed genes.

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Published in Mol Endocrinol on July 22, 2014

Authors

Michael J Guertin1, Xuesen Zhang, Scott A Coonrod, Gordon L Hager

Author Affiliations

1: Laboratory of Receptor Biology and Gene Expression (M.J.G., G.L.H.), National Cancer Institute, Bethesda, Maryland 20892; State Key Laboratory of Reproductive Medicine (X.Z.), Nanjing Medical University, Nanjing 210029, China; and Baker Institute for Animal Health (X.Z., S.A.C.), College of Veterinary Medicine, Cornell University, Ithaca, New York 14853.

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