Scaffold attachment factor A (SAF-A) and Ku temporally regulate repair of radiation-induced clustered genome lesions.

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Published in Oncotarget on June 09, 2016

Authors

Muralidhar L Hegde1,2,3, Arijit Dutta1,4, Chunying Yang1, Anil K Mantha4,5, Pavana M Hegde1, Arvind Pandey1, Shiladitya Sengupta1,3, Yaping Yu6, Patrick Calsou7, David Chen8, Susan P Lees-Miller6, Sankar Mitra1,3,4

Author Affiliations

1: Department of Radiation Oncology, Houston Methodist Research Institute, Houston, TX, USA.
2: Houston Methodist Neurological Institute, Houston, TX, USA.
3: Weill Medical College of Cornell University, Ithaca, NY, USA.
4: Department of Biochemistry and Molecular Biology, University of Texas Medical Branch, Galveston, TX , USA.
5: Center for Animal Sciences, School of Basic and Applied Sciences, Central University of Punjab, Bathinda, Punjab, India.
6: Department of Biochemistry & Molecular Biology, University of Calgary, Calgary, Canada.
7: Institut de Pharmacologie et de Biologie Structurale, CNRS, Université de Toulouse-Université Paul Sabatier, Equipe Labellisée Ligue contre le Cancer, Toulouse, France.
8: UT Southwestern Medical Center, Dallas, TX, USA.

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