Dysfunctionally phosphorylated type 1 insulin receptor substrate in neural-derived blood exosomes of preclinical Alzheimer's disease.

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Published in FASEB J on October 23, 2014

Authors

Dimitrios Kapogiannis1, Adam Boxer2, Janice B Schwartz3, Erin L Abner4, Arya Biragyn1, Umesh Masharani5, Lynda Frassetto5, Ronald C Petersen6, Bruce L Miller2, Edward J Goetzl7

Author Affiliations

1: Intramural Research Program, National Institute on Aging, Baltimore, Maryland, USA;
2: Memory and Aging Center, Department of Neurology, and.
3: Jewish Home of San Francisco, San Francisco, California, USA; Department of Medicine, University of California-San Francisco Medical Center, San Francisco, California, USA;
4: Sanders-Brown Center on Aging, University of Kentucky, Lexington, Kentucky, USA; and.
5: Jewish Home of San Francisco, San Francisco, California, USA;
6: Mayo Clinic, Rochester, Minnesota, USA.
7: Intramural Research Program, National Institute on Aging, Baltimore, Maryland, USA; Jewish Home of San Francisco, San Francisco, California, USA; Department of Medicine, University of California-San Francisco Medical Center, San Francisco, California, USA; edward.goetzl@ucsf.edu.

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