CaMKII-dependent phosphorylation of RyR2 promotes targetable pathological RyR2 conformational shift.

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Published in J Mol Cell Cardiol on June 16, 2016

Authors

Hitoshi Uchinoumi1, Yi Yang1, Tetsuro Oda1,2, Na Li3,4, Katherina M Alsina3,4,5, Jose L Puglisi1, Ye Chen-Izu1, Razvan L Cornea6, Xander H T Wehrens3,4,7,8, Donald M Bers1

Author Affiliations

1: Department of Pharmacology University of California, Davis, CA.
2: Department of Medicine and Clinical Science, Division of Cardiology, Yamaguchi University Graduate School of Medicine, Ube, Yamaguchi.
3: Cardiovascular Research Institute, Baylor College of Medicine, Houston, TX, USA.
4: Department of Molecular Physiology & Biophysics, Baylor College of Medicine, Houston, TX, USA.
5: Integrative Molecular & Biomedical Sciences Program, Baylor College of Medicine, Houston, TX, USA.
6: Department of Biochemistry, Molecular Biology and Biophysics, University of Minnesota, Minneapolis, MN.
7: Department of Medicine (Cardiology), Baylor College of Medicine, Houston, TX, USA.
8: Pediatrics, Baylor College of Medicine, Houston, TX, USA.

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